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<title>Weitere wissenschaftliche Publikationen</title>
<link>https://refubium.fu-berlin.de/handle/fub188/2</link>
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<rdf:li rdf:resource="https://refubium.fu-berlin.de/handle/fub188/27977"/>
<rdf:li rdf:resource="https://refubium.fu-berlin.de/handle/fub188/33607"/>
<rdf:li rdf:resource="https://refubium.fu-berlin.de/handle/fub188/37783"/>
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<dc:date>2026-04-18T00:39:26Z</dc:date>
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<item rdf:about="https://refubium.fu-berlin.de/handle/fub188/27977">
<title>(009) A proposal to solve a paradox when neotypifying names of fossil‐taxa</title>
<link>https://refubium.fu-berlin.de/handle/fub188/27977</link>
<description>(009) A proposal to solve a paradox when neotypifying names of fossil‐taxa
Gravendyck, Julia; Bachelier, Julien B.; Kürschner, Wolfram M.; Herendeen, Patrick S.
History has given us too many examples where types of the names of taxa have been lost. For example, the destruction of the Berlin Herbarium (B) after a bombing raid in 1943 was a severe blow for the botanical world. A major part of one of the world's largest collections and most extensive neotropical type collection was damaged or destroyed, together with many type specimens from all over the world that were on loan in Berlin at the time. Even today, unfortunate events can cause the loss of scientifically valuable type specimens.
</description>
<dc:date>2020-01-01T00:00:00Z</dc:date>
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<item rdf:about="https://refubium.fu-berlin.de/handle/fub188/33607">
<title>1,2,3, some inductive real sequences and a beautiful algebraic pattern</title>
<link>https://refubium.fu-berlin.de/handle/fub188/33607</link>
<description>1,2,3, some inductive real sequences and a beautiful algebraic pattern
Abdymanapov, Sarsengali Abdygalievich; Altynbek, Serik; Begehr, Anton; Begehr, Heinrich
By rewriting the relation 1+2=3 as √12+√22=√32 , a right triangle is looked at. Some geometrical observations in connection with plane parqueting lead to an inductive sequence of right triangles with √12+√22=√32 as initial one converging to the segment [0,1] of the real line. The sequence of their hypotenuses forms a sequence of real numbers which initiates some beautiful algebraic patterns. They are determined through some recurrence relations which are proper for being evaluated with computer algebra.
</description>
<dc:date>2021-01-01T00:00:00Z</dc:date>
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<item rdf:about="https://refubium.fu-berlin.de/handle/fub188/37783">
<title>1,2-13C2-Glucose Tracing Approach to Assess Metabolic Alterations of Human Monocytes under Neuroinflammatory Conditions</title>
<link>https://refubium.fu-berlin.de/handle/fub188/37783</link>
<description>1,2-13C2-Glucose Tracing Approach to Assess Metabolic Alterations of Human Monocytes under Neuroinflammatory Conditions
Giacomello, Ginevra; Otto, Carolin; Priller, Josef; Ruprecht, Klemens; Böttcher, Chotima; Parr, Maria Kristina
Neuroinflammation is one of the common features in most neurological diseases including multiple sclerosis (MScl) and neurodegenerative diseases such as Alzheimer’s disease (AD). It is associated with local brain inflammation, microglial activation, and infiltration of peripheral immune cells into cerebrospinal fluid (CSF) and the central nervous system (CNS). It has been shown that the diversity of phenotypic changes in monocytes in CSF relates to neuroinflammation. It remains to be investigated whether these phenotypic changes are associated with functional or metabolic alteration, which may give a hint to their function or changes in cell states, e.g., cell activation. In this article, we investigate whether major metabolic pathways of blood monocytes alter after exposure to CSF of healthy individuals or patients with AD or MScl. Our findings show a significant alteration of the metabolism of monocytes treated with CSF from patients and healthy donors, including higher production of citric acid and glutamine, suggesting a more active glycolysis and tricarboxylic acid (TCA) cycle and reduced production of glycine and serine. These alterations suggest metabolic reprogramming of monocytes, possibly related to the change of compartment (from blood to CSF) and/or disease-related. Moreover, the levels of serine differ between AD and MScl, suggesting different phenotypic alterations between diseases.
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<dc:date>2023-01-01T00:00:00Z</dc:date>
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<item rdf:about="https://refubium.fu-berlin.de/handle/fub188/20872">
<title>1,3-Dibromo-5,5-dimethylhydantoin as promoter for glycosylations using
thioglycosides</title>
<link>https://refubium.fu-berlin.de/handle/fub188/20872</link>
<description>1,3-Dibromo-5,5-dimethylhydantoin as promoter for glycosylations using
thioglycosides
Xu, Fei-Fei; Pereira, Claney L.; Seeberger, Peter H.
1,3-Dibromo-5,5-dimethylhydantoin (DBDMH), an inexpensive, non-toxic and
stable reagent, is a competent activator of thioglycosides for glycosidic bond
formation. Excellent yields were obtained when triflic acid (TfOH) or
trimethylsilyl trifluoromethanesulfonate (TMSOTf) were employed as co-
promoters in solution or automated glycan assembly on solid phase.
</description>
<dc:date>2017-01-01T00:00:00Z</dc:date>
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